Antisense oligonucleotide therapy in a humanized mouse model of MECP2 duplication syndrome

Antisense oligonucleotide therapy in a humanized mouse model of MECP2 duplication syndrome

Many mental incapacity issues are resulting from copy quantity variations, and, up to now, there have been no remedy choices examined for this class of ailments.¬†MECP2¬†duplication syndrome (MDS) is without doubt one of the most typical genomic rearrangements in males and outcomes from duplications spanning the methyl-CpG binding protein 2 (MECP2) gene locus. We beforehand […]